CaSR Controls Calcium
CaSR stands for calcium sensing receptor and they are mainly found in 4 different places:
- Parathyroid chief cells
- Ca binds to CaSR on chief cells
- Causing decreased synthesis of PTH
- PTH stands for parathyroid hormone which is produced by the parathyroid gland
- PTH increases resorption at the bones
- PTH increases Ca reabsorption at the kidneys
- PTH increases synthesis of 1a-hydroxylase which produces active vitamin D in the kidneys
- When Ca increases, PTH decreases
- Ca binds to CaSR on chief cells
- Proximal tubule of kidney
- Ca binds to CaSR in kidney
- Causing inhibition renal Ca reabsorption in the thick ascending limb of the loop of Henle = higher urinary Ca losses
- Impairs ability to concentrate urine = higher water losses (if hypercalcemia persists = nephrogenic diabetes insipidus, which can progress to shock!)
- Ca binds to CaSR in kidney
- Bone – osteoblasts and osteoclasts
- Ca binds to CaSR on osteoblast
- Increase osteoblast activity – increased bone building from increased Ca in blood
- Ca binds to CaSR on osteoclast
- Decrease osteoclast formation and activity
- Ca binds to CaSR on osteoblast
- Thyroid C cells
- Ca binds to CaSR on C cells of thyroid
- Causes release of calcitonin which has minor effects of Ca levels
- Increases renal Ca excretion
- Inhibits 1a-hydroxylase in kidneys
- Inhibits osteoclasts which break down bone and release Ca into serum
- Causes release of calcitonin which has minor effects of Ca levels
- Ca binds to CaSR on C cells of thyroid